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Aβ ELISA kit reference List

292-62301 Human β Amyloid(1-40) ELISA Kit Wako

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Curcumin decreases amyloid-beta peptide levels by attenuating the maturation of amyloid-beta precursor protein.

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Effect of apolipoprotein E phenotype on the association of plasma amyloid β and amyloid positron emission tomography imaging in Japan. https://www.ncbi.nlm.nih.gov/pubmed/28975146

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Decreased N-Acetyl Aspartate/Myo-Inositol Ratio in the Posterior Cingulate Cortex Shown by Magnetic Resonance Spectroscopy May Be One of the Risk Markers of Preclinical Alzheimer's Disease: A 7-Year Follow-Up Study.

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A flow cytometry–based in vitro assay reveals that formation of apolipoprotein E (ApoE)–amyloid beta complexes depends on ApoE isoform and cell type https://www.ncbi.nlm.nih.gov/pubmed/29950521

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REST and Neural Gene Network Dysregulation in iPSC Models of Alzheimer’s Disease https://www.ncbi.nlm.nih.gov/pubmed/30699343

298-64601 Human βAmyloid(1-40)ELISA Kit Wako Ⅱ

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Effect of apolipoprotein E phenotype on the association of plasma amyloid β and amyloid positron emission tomography imaging in Japan. https://www.ncbi.nlm.nih.gov/pubmed/28975146

294-62501 Human/Rat β Amyloid(40)ELISA Kit Wako

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Multifunction of myricetin on Aβ: neuroprotection via a conformational change of Aβ and reduction of Aβ via the interference of secretases

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Amyloid-β production via cleavage of amyloid-β protein precursor is modulated by cell density. https://www.ncbi.nlm.nih.gov/pubmed/20847415

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 Curcumin decreases amyloid-beta peptide levels by attenuating the maturation of amyloid-beta precursor protein.

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Anthocyanin-enriched bilberry and blackcurrant extracts modulate amyloid precursor protein processing and alleviate behavioral abnormalities in the APP/PS1 mouse model of Alzheimer's disease

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The retinoic acid receptor agonist Am80 increases hippocampal ADAM10 in aged SAMP8 mice https://www.ncbi.nlm.nih.gov/pubmed/23624141

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Amyloid precursor protein α- and β-cleaved ectodomains exert opposing control of cholesterol homeostasis via SREBP2.

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Decreased CALM expression reduces Aβ42 to total Aβ ratio through clathrin-mediated endocytosis of γ-secretase

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KB-R7943, an inhibitor of the reverse Na+/Ca2+ exchanger, does not modify secondary pathology in the thalamus following focal cerebral stroke in rats https://www.ncbi.nlm.nih.gov/pubmed/25123443

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 Genetic variation in δ-opioid receptor associates with increased β-and γ-secretase activity in the late stages of Alzheimer's disease

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 Mitofusin-2 knockdown increases ER–mitochondria contact and decreases amyloid β‐ peptide production

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Expression of B4GALNT1, an essential glycosyltransferase for the synthesis of complex gangliosides, suppresses BACE1 degradation and modulates APP processing https://www.ncbi.nlm.nih.gov/pubmed/27687691

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The polarity protein Par3 regulates APP trafficking and processing through the endocytic adaptor protein Numb

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Increased Spontaneous Central Bleeding and Cognition Impairment in APP/PS1 Mice with Poorly Controlled Diabetes Mellitus.

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An antioxidant specifically targeting mitochondria delays progression of Alzheimer's disease-like pathology.

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Amyloid-beta (Aβ) D7H mutation increases oligomeric Aβ42 and alters properties of Aβ-zinc/copper assemblies

https://www.ncbi.nlm.nih.gov/pubmed/22558227

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Effect of apolipoprotein E phenotype on the association of plasma amyloid β and amyloid positron emission tomography imaging in Japan. https://www.ncbi.nlm.nih.gov/pubmed/28975146

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A protocol for quantitative analysis of murine and human amyloid-β1-40 and 1-42. https://www.ncbi.nlm.nih.gov/pubmed/28768163

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 Antioxidant SkQ1 Alleviates Signs of Alzheimer's Disease-like Pathology in Old OXYS Rats by Reversing Mitochondrial Deterioration

https://www.ncbi.nlm.nih.gov/pubmed/28637402

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Long-Term Mangiferin Extract Treatment Improves Central Pathology and Cognitive Deficits in APP/PS1 Mice.

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Progressive Neuronal Pathology and Synaptic Loss Induced by Prediabetes and Type 2 Diabetes in a Mouse Model of Alzheimer's Disease.

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Activation of γ-Secretase Trimming Activity by Topological Changes of Transmembrane Domain 1 of Presenilin 1.

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Loss of kallikrein‐related peptidase 7 exacerbates amyloid pathology in Alzheimer's disease model mice

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Introduction of pathogenic mutations into the mouse Psen1 gene by Base Editor and Target-AID https://www.ncbi.nlm.nih.gov/pubmed/30042426

294-64701 Human/Rat βAmyloid(40)ELISA Kit Wako Ⅱ

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Oral administration of synthetic retinoid Am80 (Tamibarotene) decreases brain beta-amyloid peptides in APP23 mice.

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Amyloid deposition and influx transporter expression at the blood-brain barrier increase in normal aging.

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Analysis of microdissected human neurons by a sensitive ELISA reveals a correlation between elevated intracellular concentrations of Aβ42 and Alzheimer’s disease neuropathology https://www.ncbi.nlm.nih.gov/pubmed/20198479

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Isoflurane-induced spatial memory impairment by a mechanism independent of amyloid-beta levels and tau protein phosphorylation changes in aged rats https://www.ncbi.nlm.nih.gov/pubmed/22196855

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Intracerebral microinjection of interleukin-4/interleukin-13 reduces β-amyloid accumulation in the ipsilateral side and improves cognitive deficits in young amyloid precursor protein 23 mice https://www.ncbi.nlm.nih.gov/pubmed/22342341

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Temporal course of cerebrospinal fluid dynamics and amyloid accumulation in the aging rat brain from three to thirty months

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Regulation of β-amyloid level in the brain of rats with cerebrovascular hypoperfusion. https://www.ncbi.nlm.nih.gov/pubmed/21813211

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Lysosomal dysfunction in a mouse model of Sandhoff disease leads to accumulation of ganglioside-bound amyloid-β peptide.

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 Effects of long-term moderate ethanol and cholesterol on cognition, cholinergic neurons, inflammation, and vascular impairment in rats. https://www.ncbi.nlm.nih.gov/pubmed/22244974

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Effects of oxidative stress on amyloid precursor protein processing in rat and human platelets https://www.ncbi.nlm.nih.gov/pubmed/22385218

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MicroRNA-195 protects against dementia induced by chronic brain hypoperfusion via its anti-amyloidogenic effect in rats.

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β-Secretase (BACE1) inhibitors with high in vivo efficacy suitable for clinical evaluation in Alzheimer's disease.

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Cargo Delivery into the Brain by in vivo identified Transport Peptides. https://www.ncbi.nlm.nih.gov/pubmed/26411801

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